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Monitoring brain multiparameters and hypothermia in severe traumatic brain injury

Authors Vasconcelos FR, Andrade AF, Teixeira MJ, Paiva WS

Received 21 September 2016

Accepted for publication 21 September 2016

Published 8 March 2017 Volume 2017:13 Pages 721—722

DOI https://doi.org/10.2147/NDT.S122854

Checked for plagiarism Yes

Editor who approved publication: Dr Roger Pinder


Fernando Roberto de Vasconcelos, Almir Ferreira de Andrade, Manoel Jacobsen Teixeira, Wellingson Silva Paiva

Division of Neurological Surgery, Department of Neurology, University of São Paulo Medical School, São Paulo, Brazil

We read with great interest the recent study by Sun et al1 published in Neuropsychiatric Disease and Treatment. Intracranial hypertension (ICH) remains the most common cause of death in patients with traumatic brain injury (TBI).1 After the trauma, secondary injuries arise due to perfusion and molecular changes that begin after the moment of the trauma resulting from the interaction of intracerebral and extracerebral factors, which add to the neuronal injury. Hypotension, hypoglycemia, hypercapnia, respiratory hypoxia, anemic hypoxia, and electrolyte disturbances are the main factors associated with secondary injury, which can lead to ICH and cerebral hypoxia, thereby worsening the morbidity and death rates associated with brain injury.1,2 In this paper, the authors have discussed the multimodality intracranial monitoring carried out in 62 severe TBI patients, with evidence of hypothermia as a therapeutic strategy to adequately control intracranial pressure (ICP).

View the original paper by Sun and colleagues.

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