-
International Journal of Chronic Obstructive Pulmonary Disease
-
About Dovepress
Open access peer-reviewed scientific and medical journals.
-
Open Access
Dove Medical Press is now a member of the Open Access Initiative
-
An Author's Guide
A guide to help authors get their paper published.
-
Advocacy
Support Open Access and Dove Press
-
Reprints
Promotional Article Monitoring - further details
-
Favored Author Program
Real benefits for authors, including fast-track processing of papers.
Assessment of airway inflammation using sputum, BAL, and endobronchial biopsies in current and ex-smokers with established COPD
Original Research
(1991) Views (605) Full article downloads
Authors: Yudong Wen, David W Reid, Dongcheng Zhang, et al
Published Date September 2010
Volume 2010:5 Pages 327 - 334
DOI: http://dx.doi.org/10.2147/COPD.S11343
Yudong Wen, David W Reid, Dongcheng Zhang, Chris Ward, Richard Wood-Baker, E Haydn WaltersRespiratory Research Group, Menzies Research Institute, University of Tasmania, Hobart, Tasmania 7001, Australia
Rationale: Smoking effects on physiological and gross pathology in chronic obstructive pulmonary disease (COPD) are relatively well described. However, there is little known in COPD about the detailed interrelationships between lung function and inflammatory profiles in different airway compartments from the same individual and whether airway inflammation in these different compartments differs in ex- and current smokers with established COPD.
Objectives: We compared sputum, bronchoalveolar (BAL), and airway wall inflammatory profiles in current versus ex-smokers and related this to smoking intensity and lung function in 17 current and 17 ex-smokers with mild to moderate COPD.
Results: Current smokers had more sputum mast cells (% differential and absolute numbers), whereas ex-smokers had increased sputum neutrophils. In BAL, there was a significant increase in eosinophils in current smokers, but ex-smokers had significantly increased neutrophils, lymphocytes, and epithelial cells. There were no cell profile differences observed in airway biopsies between current and ex-smokers and there were no correlations between the individual inflammatory cell populations in any of the airway compartments. In current smokers only, smoking intensity was negatively correlated with lung function, and associated with a reduction in overall cellularity of both sputum and BAL.
Conclusion: Airway inflammation persists in ex-smokers with COPD, but differs from COPD current smokers. The impact of smoking appears to vary in different airway compartments and any direct relationships between cellularity and lung function tended to be negative, ie, worse lung function indicated the presence of fewer cells.
Keywords: current smokers, ex-smokers, airway cellularity, sputum, BAL, endobronchial biopsies
Readers of this article also read:
Changes in six-minute walking distance during pulmonary rehabilitation in patients with COPD and in healthy subjects
COPD and cognitive impairment: the role of hypoxemia and oxygen therapy
Tiotropium bromide inhibits TGF-ß-induced MMP production from lung fibroblasts by interfering with Smad and MAPK pathways in vitro
Pulmonary function tests, sputum induction, and bronchial provocation tests: diagnostic tools in the challenge of distinguishing asthma and COPD phenotypes in clinical practice
COPD is frequent in conditions of comorbidity in patients treated with various diseases in a university hospital
Interleukin-6 and airflow limitation in chemical warfare patients with chronic obstructive pulmonary disease
Budesonide/formoterol combination in COPD: a US perspective
Development and implementation of treadmill exercise testing protocols in COPD
Association between lung function and exacerbation frequency in patients with COPD
- Journal Indexing
See where all the Dove Press journals are indexed
- Testimonials
"You do a tremendous job!!" Ruben Restrepo, The University of Texas Health Science Center at San Antonio
- Clinical effectiveness of the Respimat® inhaler device in managing chronic obstructive pulmonary disease: evidence when compared with other handheld inhaler devices
- Chronic obstructive pulmonary disease as an independent risk factor for cardiovascular morbidity
- The pathophysiology of bronchiectasis
- Exacerbation rate, health status and mortality in COPD – a review of potential interventions




